Male LUTD may initiate in the fetal environment – decades earlier than previously believed!

Research from investigators at the UW-Madison O’Brien Center describes a new mechanism of benign prostate disease, initiated by fetal chemical exposure, which durably increases prostatic noradrenergic axon density and causes smooth muscle hyperactivity and urinary voiding dysfunction. Read the full publication here: https://pubmed.ncbi.nlm.nih.gov/34164643/

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